Rethinking Cancer: Why the Classic Theory May Be Missing Something 

Dr. C. Siddoo-Atwal, (Dr. Moondust) President and Primary Biochemist of Moondust Cosmetics Ltd., spoke at the International Conference on Cancer Science & Research, which took place November 17–19, 2025, in Singapore. Her presentation, “Is Cancer Theory Incomplete?”, invited researchers and clinicians to take a fresh look at how cancer begins—and how it might be prevented.

For decades, the dominant model of cancer focused on DNA mutations: damage leads to genetic errors, which drive uncontrolled cell growth. But as Dr. Moondust points out, many carcinogens—including UV light, microwaves, and fungal toxins—don’t always cause mutations in standard lab tests. Yet, they still cause cancer. 

Her work proposes a new perspective centered on apoptosis, the body’s natural process for removing damaged cells. In what she calls the Two-Step Model, cancer may begin not with mutations, but with repeated cycles of abnormal cell death followed by resistance to apoptosis. 

Over time, cells that no longer respond to “self-destruct” signals survive when they shouldn’t—lay the groundwork for tumor formation. These early, stress-induced changes are often epigenetic (affecting how genes behave rather than altering the DNA itself) and may even be reversible. 

Dr. Siddoo-Atwal’s findings also highlight the potential of natural compounds, such as resveratrol from grape skins, to help restore healthy apoptotic balance. In early applications, topical resveratrol reduced abnormal skin growth and supported tissue repair, suggesting that early intervention may prevent cancer progression. 

This emerging model reframes how we think about cancer’s first steps—pointing toward prevention strategies that protect cell health long before mutations take hold. 

Main References for this presentation to the International Conference on Cancer Science & Research 
📅 Singapore | November 17–19, 2025:  https://cancer.miconferences.com/speaker/chanda-siddoo-atwal